What Accessories Say About People Essay Example for Free

What Accessories Say About People Essay â€Å"Our appearance can be an important factor in developing a sense of our identity†. Nowadays, appearances are everything, and the way you dress, the car you drive, and the phone you carry tell others a great deal about yourself. First of all, the clothes you put on and the hairstyle you have can reveal a lot about you. Our clothes and hairstyles say about you far more than you think since they tend to give a general idea about your beliefs, and a little bit about your taste in music. One example of the common clothing personalities is when a person wears colorful clothes, with peace signs printed all over them, and has dreadlocks, with maybe a short messy beard, then it is obvious that he is a hippie, and these people are usually vegetarians. Another example is when you see someone dressed in black from head to toe, with some body piercings and an extreme hairstyle, then this clearly indicates that this individual is into rock music, attends rock concerts, and might even be in a rock band himself. Also, when a man wears a suit and a tie, has a shaved face and a clean haircut, it tells us that he’s either a businessman or a manager. The outer appearance can also clothes, and have medium to long beards with a short mustache. Secondly, the car you drive reflects your financial and social status. Since cars prices range from low to extremely high, it basically covers most of the classes of society, so the kind of car you own shows where you stand in society. For example, businessmen and managers tend to ride in fancy black cars. Also, when a person drives a luxurious exotic car, it goes without saying that this person comes from a wealthy family, and might even hold a notable social status. Political figures like presidents and ambassadors drive their cars around in parades. And another example is mini cars drivers. Those people are usually not showy (unless the mini car is very expensive, like a mini cooper) and some of them are practical, since they get mini cars to reduce fuel consumption. More importantly, when you find the inside of the car disorganized with lots of paper and bottles lying everywhere while it is clean from the outside, then this clearly shows that its owner is all about the looks and only cares about showing off in front of others. Finally, cell phones. Cell phones nowadays are not just an accessory; they send major messages about you and your personality. A study was carried out by TalkTalk Mobile and examined 2,000 users of the currently top three devices to see if the chosen cell phone really is and extension to their personality. The results showed that iPhone users care more about their appearance and outer-image, and are more conceited than BlackBerry or Android users. iPhone users most likely work in media, retail, and they are the most active on social media sites. While BlackBerry users came in as the busiest group of the three, with the largest number of messages and emails sent, also they are more likely to have better relationships and better-paid jobs. They mostly work in health and finance. Android users on the other hand are better mannered than the other two, although they were found as the group to watch TV and drink alcohol the most. They tend to have jobs in engineering and the government. So overall, if you pay attention to those details in others like their clothes, cars and cell phones, you will get a general idea about them and their personality.

Peter Kropotkin :: essays research papers fc

Peter Kropotkin Peter Kropotkin was a major anarchist thinker of the 19th century. His ideals have spread worldwide and have greatly influenced many of today’s political structures. His passionate vision has been a major influence many controversial wars and political debates around the globe. He was a fearless revolutionary with an intense desire for change. Kropotkin’s strong example is one we should all make an effort to follow as we are now approaching new changes within the 21st century. Kropotkin’s Life   Ã‚  Ã‚  Ã‚  Ã‚  While researching further on Kropotkin’s life, I discovered that he was not exactly raised as I would expect from such a radical anarchist thinker. In fact, I gained more respect for him when I learned that he was born into a noble family and had the willingness to give up his riches in search for his own truth. I found some interesting facts about his life in Kropotkin – the Master, by Herbert Read.   Ã‚  Ã‚  Ã‚  Ã‚  Peter Kropotkin was born in Moscow where he was the medieval Grand Prince of Kiev. He owned nearly twelve hundred male serfs in three different providences, housed about fifty servants in Moscow, and twenty-five more out in the country. He was a good master to his people and had the tendency even as a young boy to persistently fight for the less fortunate. When he was fifteen he entered the Corps of ages at St. Petersburg, a military academy consisting of only select noble children. After graduating from St. Petersburg, he became an officer in Siberia and was the elected secretary for both the reform of the prisons, and for preparing a scheme of municipal self-government. In Siberia and was brought into contact with many different social characters. He became quite rebellious through his interactions and resigned from the army in 1872 to become a geographer and anarchist carrying his extreme philosophies through Russia. He was eventually imprisoned in Russia and s oon escaped to Western Europe where he began a publication called, Le Revolte, until he was imprisoned again in France around 1882. They released him in 1885 after many protests from writers, scientists, and philosophers. He then spent about thirty years writing many books including, The Conquest of Bread, Mutual Aid, Memoirs of a Revolutionist, and Fields, Factories and workshops, during which he lived in the Hull House in Chicago and eventually moved back to Western Europe. Kropotkin's Utopian Ideals   Ã‚  Ã‚  Ã‚  Ã‚  Kropotkin had an articulate understanding of the role of law and authority within civilization.

Special Situations In Trauma Recognition Health And Social Care Essay

The earliest recordings of Crush syndrome, which is besides known as Traumatic rhabdomyolysis or Baywaters ‘ syndrome are from 1910, when German writers described symptoms of hurting, failing and brown-coloured piss in soldiers rescued after being buried under structural dust [ 1 ] . The status was decently classified in 1941 during the London blitz in patients showing with symptoms of daze, conceited appendages, tea coloured urine and nephritic failure, by British nephrologist Eric Bywater [ 1 ] . In more recent times, instances of crush syndrome have been seen in greatest Numberss following natural catastrophes such as temblors. Clinical profiling of the types of hurts treated in local infirmaries following a natural catastrophe give an indicant of the prevalence of this type of hurt in such state of affairss. Following the Wenchuan temblor in China in 2008, a sum of 19 out of 205 patients admitted to two local learning infirmaries were enduring from crush syndrome [ 2 ] , while the instances of crush syndrome reported following the 1999 Marmara temblor in Turkey were every bit high as 17 % of reported hurts [ 3 ] . While the Numberss may change, one thing that is evident is that crush syndrome is a really existent hurt menace in such state of affairss, and that acknowledgment of the marks of this hurt, and an apprehension of the right intervention methods may good ensue in lives being saved in such scenarios.Crush syndrome – Mechanism of actionCrush syndrome occurs as the consequence of a localized crush hurt. As a consequence of compaction of the musculus mass, the musculus fibers are deprived of blood and can go ischemic. With drawn-out ischaemia of greater than 6 hours in continuance, tissue mortification occurs. Direct hurt signifier the suppression force besides causes harm to the cell membrane, which consequences in the gap of intracellular Na and Ca channels with a attendant inflow of Na and Ca into the cell. This influx consequences in increased harm to the musculus fibers and accordingly exacerbates cell membrane disfunction and causes the release of ATP-inhibiting nucleases, farther damaging the cells. With the return of circulation to the ischaemic tissue country following extraction or deliverance, comes farther tissue harm. Tissue reperfusion consequences in an addition in neutrophil activity and the release of free groups. The reaction of H peroxide and the anionic signifier of O ( superoxide ) , consequences in the formation of hydroxyl groups in big concentrations which further amendss cellular constituents and causes lipid peroxidation. The attendant lipid peroxidation leads to the devastation of the cell membrane and cell lysis ( reperfusion syndrome ) .Acute kidney hurtThe major effect of rhabdomyolysis is acute kidney hurt, which if left untreated can take to kidney failure. Acute kidney injure can happen as a effect of: Nephritic vasoconstriction Tubular obstructor due to the formation of dramatis personaes Direct heme induced cytotoxicity Myoglobin is an iron-containing protein found in skeletal musculus that is structurally similar to haemoglobin, but binds one instead than four molecules of O. Its concentration is greatest in musculuss specialised for sustained contraction, where myoglobin may supply O when blood flow is cut off due to its greater affinity for O. Like hemoglobin, myoglobin is filtered through the glomeruli of the kidneys and reabsorbed in the proximal convoluted tubules by endocytosis. Within lysosomes in the cells of the tubules, the hematohiston concatenation offprints from the Fe incorporating part of the molecule ( ferrihemate ) , the free Fe is so converted to ferritin. The big volumes of myoglobin delivered to the tubule cells overwhelm the cells ability to change over Fe to ferritin. The effect of this is a build-up intracellular ferrihemate. Oxidative emphasis harm to the cannular cells consequences from the coevals of free groups by the Fe molecules. The inordinate degrees of Fe in the cannular cells, causes their resorption to be inhibited. The concentration of Fe in the piss is further increased due to resorption of the H2O in the tubules due to systemic vasoconstriction and hypovolaemia [ 4 ] . The increased concentrations of Fe in the urine consequence in the formation of dramatis personaes which cause obstructor of the nephritic tubules. The nephritic tubule obstructor by the myoglobin dramatis personaes, the free extremist formation and the vasoconstriction and hypoxia caused by hypovolaemia are the chief causes of acute nephritic failure.Alternate causes of rhabdomyolysisWhile crush syndrome is a major cause of rhabdomyolysis in comparatively important Numberss, it is non the lone cause of this hurt. There are legion other possible causes, that, while rare, are no less life endangering. These include: Other causes of musculus hurt such as electric dazes and ictuss Certain toxins such as ecstasy and cocaine Cholesterol take downing drugs such as lipid-lowering medicines and fibrates Certain viral ( Epstein Barr virus, HIV ) and bacterial ( Legionella ) infections Certain familial familial upsets such as Carnitine palmitoyltransferase 2 lack and McArdle ‘s disease Electrolyte lacks such as hypophosphatemia and hypokalaemiaDiagnosis of rhabdomyolysisThe most dependable trial for the diagnosing of rhabdomyolysis is a step of plasma creatinine kinase degrees. The enzyme is released into the blood following cell rupture and the concentration of the enzyme in the blood is relative to the extent of musculus hurt. Blood degrees more than five times above the upper bound of normal are a definite index of the presence of rhabdomyolysis. The presence of urinary myoglobin is another index of rhabdomyolysis. High go arounding concentrations of myoglobin will do myoglobin carbamide, which will ensue in a chocolate-brown stain of the patient ‘s piss. Standard urine dipstick trials detect heme and are unable to distinguish between hematurias, haemoglobinurea and myoglobin carbamide, and as such can non be used as a definite index of rhabdomyolysis. The absence of myoglobinurea can non be used to govern out a diagnosing of rhabdomyolysis either, as myoglobin is quickly metabolised by the liver. Elevated degrees of lactate dehydrogenase and aspartate aminotransferase in the blood are other utile indexs of the presence of rhabdomyolysis.TreatmentThe first intervention measure should be the remotion of the oppressing force, nevertheless if the victim has been trapped for an drawn-out period of clip, remotion of the force may ensue in reperfusion syndrome which may ensue in sudden decease. Initial medical intervention, whether in a infirmary scene or in the field, should get down with aggressive rehydration of the patient. Treatment guidelines from the U.S. Department of Health and Human Services recommend the disposal of 1500 milliliter of endovenous fluids per hr, with disposal of endovenous fluids with Osmitrol to keep diuresis at 300 milliliters per hr, with the purpose of forestalling nephritic failure [ 5 ] . The same guidelines recommend that in the instance of intervention in a pre-hospital scene, disposal of endovenous fluids should get down prior to the realease of the crushed portion [ 5 ] . During the first 24 hours of intervention, up to 12 liters of endovenous fluids should be administered. [ 6 ] . Other fluid intervention regimens recommend the disposal of 1 liter of isosmotic saline with 1 liters of 5 % dextroglucose to which 100 mmol of Na hydrogen carbonate has been added [ 7 ] . The purpose of this intervention is to rectify metabolic acidosis in add-on to rinsing the myoglobin out of the patients system and cut downing the formation of dramatis personaes. In add-on to the remotion of myoglobin and dramatis personaes from the patients system, endovenous fluid disposal besides aims at rectifying possible electrolyte upsets. If initial fluid intervention does non turn out effectual, dialysis intervention may be required [ 7 ] . With prompt and right medical intervention, the endurance rate for rhabdomyolysis induced acute nephritic failure is near to 80 % [ 8 ] .DecisionAlthough natural catastrophes remain rare in Western Europe, there remains the possibility the crush syndrome may happen in any catastrophe state of affairs such as route clangs or detonations. While crush syndrome and the potency for acute nephritic failure triggered by rhabdomyolysis carry a terrible menace of mortality, prompt and right intervention has the possible to salvage the lives of the bulk of crush syndrome sick persons.

Higher Education And The Consequences Of The Rising...

Presidential candidates are predicting the next economic crisis facing our country is the rising student loan debt (Josuweit, 2015). Some refer to it as a â€Å"generational time-bomb ticking with adults under age 35 disproportionately bearing the brunt of escalating inequality† (Collins, 2015). Today’s college graduates are entering an economy with stagnant wages and a government that can no longer afford to provide a safety net. This paper will examine the economic benefits of higher education and the consequences of the rising student loan debt to the nation as a whole. Magnitude of the Student Loan Debt Crisis One in five U.S. households is affected by student loan debt. Forty-one million Americans carry student loan debt with a median†¦show more content†¦The federal direct loan program allows students to borrow a maximum of $5,500 in their first year of college, $6,500 in their second year, and $7,500 in their third and fourth years of undergraduate education (Education, Loans: subsidized and unsubsidized, n.d.). Since these amounts are far below the annual cost of approximately $23,872 to attend a four year institution (the most recent data as of 2012-2013 included tuition, fees, room and board), the financial aid office assembles a package of private loans, often with interest rates as high as 18%, compared to 6% for federal student loans (Education, Tuition costs of colleges and universities, 2013). Ninety percent of student loans other than the federal direct loans require a cosigner with good credit history (Education, Tuition costs of colleges and universities, 2013; Kaspe rkevic, 2014). As the cost of college continues to rise above the rate of inflation each year, more and more students are turning to private loans to finance their education. In the 2014-2015 academic year, lenders made $6.7 billion in new private loans, which represents 7 to 10 percent of the total debt (Neubecker, 2015). Why is Student Debt Growing? A 2012 study done by Harvard University and the University of Virginia reported a sharp rise in private-sector borrowing to finance higher education; the total debt grew from $1.5 billion (constant 2009 dollars) in 1995-96 to $21.8